MCF-7 cells growing in culture were used to of mifepristone and tamoxifen on apoptotic by an additive induction of apoptosis in both estrogen and
/8/2013Curcumin induces cell death and restores tamoxifen sensitivity in the antiestrogen-resistant breast cancer cell lines MCF-7/LCC2 anti-apoptosis
TY - JOUR. T1 - Acquired Tamoxifen Resistance in MCF-7 Breast Cancer Cells Requires Hyperactivation of eIF4F-Mediated Translation. AU - Fagan,Dedra H.
The protein expression of claudin-1 was slightly increased by treatment with 20 μM tamoxifen in MCF-7 cellular resistance to apoptosis in MCF-7
Momordica cochinchinensis Aril Extract Induces Apoptosis in Human MCF-7 Breast Cancer Cells (SERMs) such as tamoxifen have been
The MCF7/TAMR-1 cell line is a good model cell to study in tamoxifen resistant MCF-7 Mediated Apoptosis and is a Predictor of
This study was undertaken to investigate the global proteomic alterations in a tamoxifen resistant MCF-7 breast MCF -TamR cells per 100 and apoptosis of
Tamoxifen Inhibits TRPV6 Activity via Estrogen Receptor–Independent Pathways in TRPV6-Expressing MCF-7 Breast Cancer Cells
Tamoxifen induces TGF-β1 activity and The results presented in this study suggest that TAM induces apoptosis of MCF-7 cells and it may be mediated by the
Overexpression of HER2 modulates bcl-2, bcl-XL, and tamoxifen-induced apoptosis in human MCF-7 breast cancer cells.
n tamoxifen-resistant MCF- 7 (TAMR/MCF-7) cells. Methods: Cytotoxicity, apoptosis and autophagic cell death induced by SAHA were studied.
Tamoxifen (TAM) is the earliest non-steroidal antiestrogen drug, which has been widely used in endocrine therapy targeting breast cancer. The aim of the present study was to investigate the effect of TAM on the proliferation, apoptosis, migration and invasion of the estrogen‑positive (ER+) breast cancer cell line MCF‑7 in vitro, and
hat tamoxifen inhibited MCF-7 cell growth mainly by G1 arrest, while cisplatin did not cause the pronounced cell-cycle typical of apoptosis,
. Breast. 2000 Apr;9(2):96-106. Tamoxifen increases apoptosis but does not influence markers of proliferation in an MCF-7 xenograft model of breast cancer.